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We reason that in chronic obesity, sustained GLP-1 activation in Tmprss2 / mice improves insulin sensitivity and glucose metabolism but, at the same time, paradoxically regulates insulin-induced GIP secretion independent of PAR2 signaling, thus superseding TMPRSS2 deficiency

doi: 10.1126/science.aac8469

Pathogens that are administered in pregnant rodents range from viruses to bacteria and parasites, and their effects on ASD-related behavioral changes have been examined

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