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Coskun, S
Despite the link, the role of insulin abnormalities in PD remains contentious, with little difference found between patients with PD and healthy controls in peripheral insulin resistance, quantified using a hyperinsulinemic-euglycemic clamp [71]
Tissue-threshold split receptor density, downstream signal amplification, and per-tissue glucagon sensitivity differ across liver, visceral fat, sinus node, and subcutaneous fat
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