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However, in diabetic wounds, the transition from M1 to M2 is disrupted, leading to the accumulation of M1 macrophages, which produce ROS and sustain long-term inflammation

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ROS dysregulation in cardiovascular diseases ROS dysregulation represents a central node in cardiovascular diseases progression [314], involving endothelial dysfunction [315], cardiomyocyte injury [316], mitochondrial [317] and endoplasmic reticulum stress [318], vascular remodeling [319], and atherosclerosis [320]