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Rat pancreatic islets were incubated in different concentrations glucose solutions, with cinchonine or Exendin (9 39) added to determine insulin secretion levels

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[36] reported that exendin-4 treatment decreases hepatic glucose output at hyperinsulinemic states and promotes hepatic insulin signaling by potentiating tyrosine phosphorylation of the insulin receptor substrate-2 in 90% pancreatectomized diabetic rats fed high fat (40% energy from fat) diets