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Karstens et al., 2019)

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Pharmacokinetic data revealed that the 200mg nimacimab monotherapy arm, which did not meet its primary endpoint, did not achieve the serum exposure predicted by preclinical models, warranting potential further study of nimacimab at higher doses

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On one hand, elevated ROS levels can precipitate oxidative stress, leading to macromolecular damage and subsequent activation of apoptotic pathways, such as the p53-mediated tumor suppress pathway [217]