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The release of proteolytic enzymes, such as calpains, from dying hepatocytes can also mediate the progression of APAP-induced liver injury by damaging neighboring hepatocytes.8, 9 Furthermore, the rate of replenishment of cellular GSH stores after initial depletion can affect the extent of oxidative damage and alter the course of APAP-induced liver injury

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However, irrespective of whether ASV abundance was weighted in the analysis, beta diversity analyses revealed distinct clustering by treatment group

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