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Copper deficiency results in reduced transport via COX17 to SCO1/SCO2 and COX11, leading to diminished CCO synthesis (45)

The inherent limitations of animal models in fully capturing the heterogeneity and complexity of AD in human necessitate carefully designed clinical trials with well-defined patient populations and outcome measures

In a post hoc subgroup analysis by the same group, blinded qualitative scoring of the calluses at 5 weeks suggested that patients who received PTH 134 had a more rich callus formation [33]

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