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Description

These data highlight the dual action of DPP-4 inhibition, which targets both GLP-1 and GIP to improve glucose homeostasis

Specific injury sites

The increase in Akt and ERK phosphorylations lead to cardiomyocyte growth and activation of glucose metabolism [24],[39]-[41]

PMID: 28057329

A.Da SilvaT

Second, the niosomal bilayer structure enables controlled and sustained drug release, as the vesicles gradually release their cargo over an extended period rather than releasing it all at once [122,124]