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It remains to be determined whether this effect extends to other substrates and cell types

The proposed mechanism involves controlling glutamate release in the synaptic cleft, modulating NMDA receptor stimulation, affecting excitatory amino acid transporter (EAAT)-mediated reuptake, and maintaining the glutamate/glutamine cycle within astrocytes

the second option is to substitute the substance with another which does not provoke the symptoms

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Bierze udzia w ochronie grup tiolowych (SH) w biakach oraz w redukcji mostkw disiarczkowych (SS)

Uniquely hominid features of adult human astrocytes