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GLP-1 inhibits intestinal contractions through mechanisms that may involve the central nervous system via vagal pathways or direct action on central receptors [24], as well as the enteric nervous system by modulating neurotransmission through presynaptic receptors affecting nitric oxide release [25,26]

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Other HDL-induced signaling pathways that result in increased cholesterol and lipid efflux include protein kinase A (PKA) (785,786), cell division control protein 42 (Cdc42) (787), and Janus kinases-2 (JAK2) (788,789) cascades

When electrophilic stress or oxidative stress occurs, KEAP1 releases NRF2, which translocates to the nucleus and binds the ARE, upregulating NQO1 along with hundreds of other protective genes